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NSJ Bioreagents

SKU:V9506-20UG

FOXP1 Antibody, 20 ug

FOXP1 Antibody, 20 ug

Regular price $259.00 USD
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Transcriptional repressor. It plays an important role in the specification and differentiation of lung epithelium. Can act with CTBP1 to synergistically repress transcription but CTPBP1 is not essential. Essential transcriptional regulator of B cell development. FOXP1 protein plays an important role in the specification and differentiation of lung epithelium, also acts cooperatively with FOXP4 to regulate lung secretory epithelial cell fate and regeneration by restricting the goblet cell lineage program.

Specifications

Catalog No V9506-20UG
Family Primary antibody
Qty 20 ug
Formulation 0.2 mg/ml in 1X PBS with 0.1 mg/ml BSA (US sourced), 0.05% sodium azide
Format Purified
Clone rFOXP1/6902
Host Animal Mouse
Clonality Recombinant Mouse Monoclonal
Isotype Mouse IgG1, kappa
Species Reactivity Human
Application IHC-P
Application Details Immunohistochemistry (FFPE): 1-2ug/ml
Application Note Optimal dilution of the FOXP1 antibody should be determined by the researcher.
Localization Nucleus
Immunogen A portion of amino acids 350-450 was used as the immunogen for the FOXP1 antibody.
Purity Protein A/G affinity
Storage Aliquot the FOXP1 antibody and store frozen at -20oC or colder. Avoid repeated freeze-thaw cycles.
Limitation This FOXP1 antibody is available for research use only.
Uniprot # Q9H334
Status Available
PDF Link https://www.nsjbio.com/tds-pdf/foxp1-antibody-rfoxp16902-v9506
Title FOXP1 Antibody
Description Transcriptional repressor. It plays an important role in the specification and differentiation of lung epithelium. Can act with CTBP1 to synergistically repress transcription but CTPBP1 is not essential. Essential transcriptional regulator of B cell development. FOXP1 protein plays an important role in the specification and differentiation of lung epithelium, also acts cooperatively with FOXP4 to regulate lung secretory epithelial cell fate and regeneration by restricting the goblet cell lineage program.
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