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NSJ Bioreagents

SKU:V9291-20UG

Recombinant MSH2 Antibody, 20 ug

Recombinant MSH2 Antibody, 20 ug

Regular price $259.00 USD
Regular price Sale price $259.00 USD
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Mutations in DNA mismatch repair genes are associated with hereditary nonpolyposis colorectal cancer (HNPCC). Initially, inherited mutations in the MSH2 and MLH1 homologs of the bacterial DNA mismatch repair genes MutS and MutL were found at high frequency in HNPCC and were shown to be associated with microsatellite instability. The demonstration that 10 to 45% of pancreatic, gastric, breast, ovarian and small cell lung cancers also display microsatellite instability has been interpreted to suggest that DNA mismatch repair is not restricted to HNPCC tumors but is a common feature in tumor initiation or progression.

Specifications

Catalog No V9291-20UG
Family Primary antibody
Qty 20 ug
Formulation 0.2 mg/ml in 1X PBS with 0.1 mg/ml BSA (US sourced), 0.05% sodium azide
Format Purified
Clone rMSH2/6548
Host Animal Mouse
Clonality Recombinant Mouse Monoclonal
Isotype Mouse IgG2a, kappa
Species Reactivity Human, Cat, Dog
Application WB, IHC-P
Application Details Western blot: 1-2ug/ml,Immunohistochemistry (FFPE): 1-2ug/ml
Application Note Optimal dilution of the recombinant MSH2 antibody should be determined by the researcher.
Localization Nucleus
Immunogen Recombinant full-length human MSH2 protein was used as the immunogen for the recombinant MSH2 antibody.
Purity Protein A/G affinity
Storage Aliquot the recombinant MSH2 antibody and store frozen at -20oC or colder. Avoid repeated freeze-thaw cycles.
Limitation This recombinant MSH2 antibody is available for research use only.
Uniprot # IDP43246
Status Available
PDF Link https://www.nsjbio.com/tds-pdf/recombinant-msh2-antibody-rmsh26548-v9291
Title Recombinant MSH2 Antibody
Description Mutations in DNA mismatch repair genes are associated with hereditary nonpolyposis colorectal cancer (HNPCC). Initially, inherited mutations in the MSH2 and MLH1 homologs of the bacterial DNA mismatch repair genes MutS and MutL were found at high frequency in HNPCC and were shown to be associated with microsatellite instability. The demonstration that 10 to 45% of pancreatic, gastric, breast, ovarian and small cell lung cancers also display microsatellite instability has been interpreted to suggest that DNA mismatch repair is not restricted to HNPCC tumors but is a common feature in tumor initiation or progression.
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