GSK3Beta Monoclonal Antibody

The protein encoded by this gene is a serine-threonine kinase, belonging to the glycogen synthase kinase subfamily. It is involved in energy metabolism, neuronal cell development, and body pattern formation. Polymorphisms in this gene have been implicated in modifying risk of Parkinson disease, and studies in mice show that overexpression of this gene may be relevant to the pathogenesis of Alzheimer disease. Alternatively spliced transcript variants encoding different isoforms have been found for this gene.

Introducing the GSK3BETA Monoclonal Antibody, a cutting-edge scientific tool designed to revolutionize research in the field of molecular biology. This highly specific antibody is meticulously engineered to target and bind to the GSK3BETA protein, enabling researchers to gain invaluable insights into its function and regulation.

Crafted with utmost precision, the GSK3BETA Monoclonal Antibody boasts exceptional affinity and selectivity, ensuring accurate and reliable results. Its advanced formulation guarantees optimal performance across a wide range of applications, including Western blotting, immunoprecipitation, and immunofluorescence.

With its unrivaled sensitivity, this antibody allows for the detection of even the lowest levels of GSK3BETA protein expression, facilitating comprehensive investigations into its role in various cellular processes. Researchers can now delve deeper into the intricate mechanisms underlying cell signaling pathways, neurodegenerative disorders, and cancer progression.

The GSK3BETA Monoclonal Antibody is meticulously validated through rigorous quality control measures, ensuring consistent and reproducible results. Its exceptional stability and long shelf life guarantee prolonged usability, making it an indispensable asset for laboratories and research institutions worldwide.

Embrace the future of molecular biology research with the GSK3BETA Monoclonal Antibody, and unlock the secrets of cellular function with unparalleled precision and reliability.