Trk A(Phospho Tyr701) Polyclonal Antibody

This gene encodes a member of the neurotrophic tyrosine kinase receptor (NTKR) family. This kinase is a membrane-bound receptor that, upon neurotrophin binding, phosphorylates itself and members of the MAPK pathway. The presence of this kinase leads to cell differentiation and may play a role in specifying sensory neuron subtypes. Mutations in this gene have been associated with congenital insensitivity to pain, anhidrosis, self-mutilating behavior, mental retardation and cancer. Alternate transcriptional splice variants of this gene have been found, but only three have been characterized to date.

The TRK A(PHOSPHO TYR701) Polyclonal Antibody is a highly specific and sensitive tool designed for the detection and analysis of phosphorylated tyrosine 701 residue on the TRK A protein. This antibody is produced using a polyclonal approach, ensuring a diverse range of antibodies that recognize different epitopes on the target protein.

The TRK A(PHOSPHO TYR701) Polyclonal Antibody exhibits exceptional affinity and selectivity, enabling accurate and reliable detection of phosphorylated tyrosine 701 residue on TRK A. It has been extensively validated for various applications, including Western blotting, immunoprecipitation, immunofluorescence, and immunohistochemistry.

With its superior performance, this antibody allows for precise quantification and localization of phosphorylated tyrosine 701 residue on TRK A, facilitating in-depth investigations into the signaling pathways and biological functions associated with this phosphorylation event.

The TRK A(PHOSPHO TYR701) Polyclonal Antibody is manufactured under stringent quality control measures, ensuring batch-to-batch consistency and reproducibility. It is supplied in a convenient format, ready for immediate use in laboratory experiments.

Choose the TRK A(PHOSPHO TYR701) Polyclonal Antibody for its exceptional sensitivity, specificity, and versatility, and unlock new insights into the role of phosphorylated tyrosine 701 residue on TRK A in various cellular processes and disease mechanisms.