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BT Lab

SKU:BT-AP02718

FoxO1A (Acetyl Lys245) Rabbit Polyclonal Antibody

FoxO1A (Acetyl Lys245) Rabbit Polyclonal Antibody

Regular price $275.00 USD
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disease:Chromosomal aberrations involving FOXO1 are a cause of rhabdomyosarcoma 2 (RMS2)

The FoxO1A (Acetyl Lys245) Rabbit Polyclonal Antibody is a highly specific and reliable tool for the detection and analysis of FoxO1A protein in various biological samples. This antibody has been developed using advanced immunization techniques and exhibits exceptional sensitivity and specificity towards the acetylated form of FoxO1A at Lys245 residue.

FoxO1A is a transcription factor that plays a crucial role in regulating various cellular processes, including cell cycle progression, apoptosis, and glucose metabolism. The acetylation of FoxO1A at Lys245 has been shown to modulate its transcriptional activity and subcellular localization, making it an important post-translational modification for the regulation of FoxO1A-mediated signaling pathways.

The FoxO1A (Acetyl Lys245) Rabbit Polyclonal Antibody has been extensively validated for its performance in various applications, including Western blotting, immunohistochemistry, and immunofluorescence. It exhibits high affinity and specificity towards the acetylated form of FoxO1A, enabling accurate and reliable detection of this protein in complex biological samples.

This antibody is produced using high-quality rabbit polyclonal antibodies that have been carefully selected and purified to ensure optimal performance and minimal cross-reactivity. It has been validated for use in a wide range of species, including human, mouse, and rat, making it a versatile tool for researchers working in different fields of study.

In summary, the FoxO1A (Acetyl Lys245) Rabbit Polyclonal Antibody is a valuable tool for the detection and analysis of FoxO1A protein in various biological samples. Its high specificity, sensitivity, and versatility make it an ideal choice for researchers studying the role of FoxO1A in cellular processes and disease mechanisms.

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